What Physicians Are Learning From Autopsy Findings of Coronavirus (COVID-19) Patients
#coronavirus #covid19 #covid _19
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This is where the direct attack on other organs that have ACE2 receptors can happen, consisting of heart muscle, kidneys, blood vessels, liver, and the brain. That patient had meningitis.
So the virus is sometimes going to all these different organs by methods of connecting to the ACE2 receptors that exist, but that’s not even the entire story.
The alveoli’s next-door neighbor is guessed who, yeah, the tiniest blood vessels in our body, blood vessels. In one research study from the Netherlands, 1/3rd of hospitalized with COVID-19 got embolisms despite already being on prophylactic doses of blood slimmers. Not just are you getting the swelling with the cytokine storm, however you’re likewise forming blood embolisms, that can travel to other parts of the body, and cause major clogs, successfully damaging those organs.
So wait a minute doc, you’re informing me that this can trigger organ damage by.
1) Straight attacking organs by their ACE2 receptor? Yup.
2) Indirectly assaulting organs by method of civilian casualties from the cytokine storm? Yup.
3) Indirectly trigger damage to organs by methods of embolism? yup.
4) Indirectly trigger damage as a result of low oxygen levels, improper ventilator settings, drug treatments themselves, and/or all of these things integrated? Yeah.
Endothelial cells are more susceptible to dying in people with preexisting endothelial dysfunction, which is regularly associated with being a male, being a smoker, having hypertension, diabetes, and weight problems. Blood clots can form and/or travel to other parts of the body. When blood clots travel to the toes, and cause obstructions in blood circulation there, implying anemia or infarction, that can trigger gangrene there. And lots of times patients with gangrene need amputation, and “COVID toes”.
Is antiphospholipid antibody syndrome (APS), the cause of all these blood embolisms in patients with serious COVID? Perhaps. Some patients with APS have what’s called devastating APS, where these patients can have strokes, seizures, heart attacks, kidney failure, ARDS, skin changes like the ones I mentioned. Viral transmittable diseases, especially those of the respiratory system, have been reported as being the triggers for CAPS.
Various elements increase the risk of developing arterial apoplexy. Classically, the cardiovascular-dependent risk factors linked in clotting have actually been hypertension, suggesting hypertension, high levels of cholesterol, cigarette smoking, diabetes, age, chemotherapy, and degree of infection. All of these contribute towards establishing arterial apoplexy.
A lot of clients with serious COVID-19 have certain labs that resemble DIC, such as increased PT/INR, increased PTT, reduced levels of platelets. The reason why these COVID patients who established embolisms in the research study I discussed previously, the factor why they do not have DIC, is actually 2 reasons, one, they weren’t having extensive bleeding, and two, they did not have low fibrinogen levels. And if its really DIC, you would have both of those things.
Anyway, you can most likely obtain from this video why it’s so hard for doctors to find out what is going on with this infection. In between the variable methods this disease can present in various clients, and the different ways that organs can suffer damage, yeah, this is actually, truly actually, made complex.
Are BLOOD CLOTS the reason COVID19 patients are dying?
Video Link – https://youtu.be/qoJ4VDaGSfY
Dr. Mike Hansen, MD
. Crucial Care Medicine.
Website: https://doctormikehansen.com/
IG: http://instagram.com/doctor.hansen/
#coronavirus #covid19 #covid _19
https://covid19globalcases.com/what-doctors-are-knowing-from-autopsy-findings-of-coronavirus-covid-19-clients/
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